The regulatory effect of chitooligosaccharides on islet inflammation in T2D individuals after islet cell transplantation: the mechanism behind Candida albicans abundance and macrophage polarization/Experiment 3/Signature 2
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< The regulatory effect of chitooligosaccharides on islet inflammation in T2D individuals after islet cell transplantation: the mechanism behind Candida albicans abundance and macrophage polarization | The regulatory effect of chitooligosaccharides on islet inflammation in T2D individuals after islet cell transplantation: the mechanism behind Candida albicans abundance and macrophage polarization/Experiment 3
Reviewed Marked as Reviewed by Svetlana up on 2025-4-3
Source: Figure 8B and 8C
Description: Analysis of the gut fungal community abundance differences between the RA (Rapamycin alone) and RC (Rapamycin + Chitooligosaccharides) groups of HMA-T2D (Human Microbiota associated type 2 diabetes) mice by LEfSe analysis
Abundance in Group 1: decreased abundance in RC (Rapamycin + Chitooligosaccharides ) treatment group of HMA-T2D (Human Microbiota associated type 2 diabetes) mice
| NCBI | Quality Control | Links |
|---|---|---|
| Arxiozyma telluris | ||
| Candida albicans | ||
| Hanseniaspora | ||
| Hanseniaspora uvarum | ||
| Kazachstania | ||
| Mattesia | ||
| unclassified Mattesia |
Revision editor(s): Tosin
